Joseph (Joe) Lipsick
Publications
- Duplication and maintenance of the Myb genes of vertebrate animals. Biol Open. 2013; (2): 101-10
- The complex containing Drosophila Myb and RB/E2F2 regulates cytokinesis in a histone H2Av-dependent manner. Mol Cell Biol. 2013; (9): 1809-18
- Drosophila lin-52 acts in opposition to repressive components of the Myb-MuvB/dREAM complex. Mol Cell Biol. 2012; (16): 3218-27
- Epigenetic regulation of olfactory receptor gene expression by the Myb-MuvB/dREAM complex. Genes Dev. 2012; (22): 2483-98
- Animal-specific C-terminal domain links myeloblastosis oncoprotein (Myb) to an ancient repressor complex. Proc Natl Acad Sci U S A. 2011; (42): 17438-43
- MYB expression and translocation in adenoid cystic carcinomas and other salivary gland tumors with clinicopathologic correlation. Am J Surg Pathol. 2011; (1): 92-9
- The C-MYB story--is it definitive? Proc Natl Acad Sci U S A. 2010; (40): 17067-8
- A conserved acidic patch in the Myb domain is required for activation of an endogenous target gene and for chromatin binding. Mol Cancer. 2008: 77
- Epigenetic regulation of gene expression by Drosophila Myb and E2F2-RBF via the Myb-MuvB/dREAM complex. Genes Dev. 2008; (5): 601-14
- Loss of Drosophila Myb interrupts the progression of chromosome condensation. Nat Cell Biol. 2007; (5): 581-7
- v-Myb represses the transcription of Ets-2. Oncogene. 2007; (8): 1238-44
- Myb proteins inhibit fibroblast transformation by v-Rel. Mol Cancer. 2006: 54
- Functional evolution of the vertebrate Myb gene family: B-Myb, but neither A-Myb nor c-Myb, complements Drosophila Myb in hemocytes. Genetics. 2005; (1): 215-29
- synMuv verite--Myb comes into focus. Genes Dev. 2004; (23): 2837-44
- Role for a Drosophila Myb-containing protein complex in site-specific DNA replication. Nature. 2002 Dec 19-26; (6917): 833-7
- Mutation of the Drosophila homologue of the Myb protooncogene causes genomic instability. Proc Natl Acad Sci U S A. 2002; (11): 7438-43
- Mutational analysis of the transcriptional activation domains of v-Myb. Oncogene. 2002; (10): 1611-5
- BS69, an adenovirus E1A-associated protein binds to and inhibits the transcriptional activity of c-Myb Oncogene. 2001
- BS69, an adenovirus E1A-associated protein, inhibits the transcriptional activity of c-Myb. Oncogene. 2001; (1): 125-32
- Functional analysis of carboxy-terminal deletion mutants of c-Myb. J Virol. 1999; (7): 5875-86
- Myb and oncogenesis. Adv Cancer Res. 1999: 21-60
- Transcriptional activation by the myb proteins requires a specific local promoter structure. FEBS Lett. 1999; (3): 401-10
- Transcriptional regulation by the carboxyl terminus of c-Myb depends upon both the Myb DNA-binding domain and the DNA recognition site. Oncogene. 1999; (23): 3452-60
- Transformation by v-Myb. Oncogene. 1999; (19): 3047-55
- D-type cyclins repress transcriptional activation by the v-Myb but not the c-Myb DNA-binding domain. EMBO J. 1998; (1): 255-68
- Myb-related Schizosaccharomyces pombe cdc5p is structurally and functionally conserved in eukaryotes. Mol Cell Biol. 1998; (7): 4097-108
- Overexpression of an alternatively spliced form of c-Myb results in increases in transactivation and transforms avian myelomonoblasts. J Virol. 1998; (8): 6813-21
- Cells transformed by a v-Myb-estrogen receptor fusion differentiate into multinucleated giant cells. J Virol. 1997; (5): 3760-6
- Constitutive expression of full-length c-Myb transforms avian cells characteristic of both the monocytic and granulocytic lineages. Cell Growth Differ. 1997; (1): 35-45
- Myb binding sites within the N-ras promoter repress transcription. Oncogene. 1997; (2): 193-202
- FAETL motif required for leukemic transformation by v-Myb. J Virol. 1996; (8): 5600-10
- One billion years of Myb. Oncogene. 1996; (2): 223-35
- By-pass of TPA-induced differentiation and cell cycle arrest by the c-Myb DNA-binding domain. Oncogene. 1995; (4): 735-41
- Dissociation of transcriptional activation and oncogenic transformation by v-Myb. Oncogene. 1995; (9): 1771-9
- Mutations in the DNA-binding and transcriptional activation domains of v-Myb cooperate in transformation. J Virol. 1995; (4): 2515-24
- Retinoic acid receptor alpha suppresses transformation by v-myb. Mol Cell Biol. 1995; (5): 2474-81
- Weak transcriptional activation is sufficient for transformation by v-Myb. Virology. 1995; (2): 467-77
- Transformation of myelomonocytic cells by the avian myeloblastosis virus is determined by the v-myb oncogene, not by the unique long terminal repeats of the virus. J Virol. 1994; (4): 2752-5
- c-Myb prevents TPA-induced differentiation and cell death in v-Myb transformed monoblasts. Oncogene. 1994; (1): 237-45
- Dicistronic selection for nuclear proteins in living animal cells. Gene. 1993; (1): 145-9
- Differential transcriptional activation by v-myb and c-myb in animal cells and Saccharomyces cerevisiae. Mol Cell Biol. 1993; (7): 4423-31
- Individual repeats of Drosophila Myb can function in transformation by v-Myb. J Virol. 1993; (12): 7332-9
- Oncogenic truncation of the first repeat of c-Myb decreases DNA binding in vitro and in vivo. Mol Cell Biol. 1993; (12): 7334-48
- c-Myb and v-Myb are differentially phosphorylated by p42mapk in vitro. Oncogene. 1993; (8): 2259-65
- A highly conserved cysteine in the v-Myb DNA-binding domain is essential for transformation and transcriptional trans-activation. Oncogene. 1992; (5): 1005-9
- Carboxy-terminal elements of c-Myb negatively regulate transcriptional activation in cis and in trans. Genes Dev. 1992; (12B): 2524-35
- Definition of functional domains in P135gag-myb-ets and p48v-myb proteins required to maintain the response of neuroretina cells to basic fibroblast growth factor. J Virol. 1992; (1): 160-6
- Expression of the CD4 gene requires a Myb transcription factor. Mol Cell Biol. 1992; (4): 1592-604
- Protein truncation is not required for c-myb proto-oncogene activity in neuroretina cells. J Virol. 1992; (11): 6773-6
- The Myb DNA-binding domain is highly conserved in Dictyostelium discoideum. Oncogene. 1992; (3): 589-96
- Determinants of sequence-specific DNA-binding by p48v-myb. Oncogene. 1991; (2): 265-73
- Protein truncation is required for the activation of the c-myb proto-oncogene. Mol Cell Biol. 1991; (8): 3987-96
- Transformation by v-myb correlates with trans-activation of gene expression. Mol Cell Biol. 1990; (6): 2591-8
- trans activation of gene expression by v-myb. Mol Cell Biol. 1990; (5): 2285-93
- DNA-binding activity associated with the v-myb oncogene product is not sufficient for transformation. J Virol. 1988; (11): 4398-402
- Specific amino acid substitutions are not required for transformation by v-myb of avian myeloblastosis virus. J Virol. 1988; (3): 1093-6
- Structural and functional domains of the myb oncogene: requirements for nuclear transport, myeloid transformation, and colony formation. J Virol. 1988; (6): 1981-8
- c-myb protein expression is a late event during T-lymphocyte activation. Mol Cell Biol. 1987; (9): 3358-60
- env-encoded residues are not required for transformation by p48v-myb. J Virol. 1987; (3): 933-6
- v-myb does not prevent the expression of c-myb in avian erythroblasts. J Virol. 1987; (10): 3284-7
- Antibodies to the evolutionarily conserved amino-terminal region of the v-myb-encoded protein detect the c-myb protein in widely divergent metazoan species. Proc Natl Acad Sci U S A. 1986; (13): 4685-9
- Expression of molecular clones of v-myb in avian and mammalian cells independently of transformation. J Virol. 1986; (2): 267-75
- The myb oncogene. Gene Amplif Anal. 1986: 73-98
- Biologically active proviral clone of myeloblastosis-associated virus type 1: implications for the genesis of avian myeloblastosis virus. J Virol. 1985; (1): 240-4
- Cryptococcal myocarditis in acquired immune deficiency syndrome. Am J Cardiol. 1985; (9): 1240
- Avian myeloblastosis virus and E26 virus oncogene products are nuclear proteins. Proc Natl Acad Sci U S A. 1984; (14): 4265-9
- Identification of the leukemogenic protein of avian myeloblastosis virus and of its normal cellular homologue. Proc Natl Acad Sci U S A. 1983; (10): 2834-8
- Enteric adenoviruses: detection, replication, and significance. J Clin Microbiol. 1979; (4): 574-8
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