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Tushar Desai

Academic Appointments

  • Assistant Professor of Medicine (Pulmonary and Critical Care)

Key Documents

Contact Information

  • Clinical Offices
    Chest Clinic 300 Pasteur Dr A283 MC 5351 Stanford, CA 94305
    Tel Work (650) 725-7061 Fax (650) 498-6288


Clinical Focus

  • Pulmonary Disease
  • Interstitial Lung Diseases

Academic Appointments

Honors and Awards

  • Interrogation of Individual Cells to Identify Progenitor Cells and Their Niches (U01), NIH/NHLBI (2014-2016)
  • Development and Maintenance of the Alveolar Type 1 Cell (K08), NIH/NHLBI (2008-2013)
  • Fellowship in Pulmonary Research, Parker B. Francis Foundation (2006-2009)
  • Retinoic Acid in Early Lung Morphogenesis, GlaxoSmithKline Pulmonary Fellowship Award (2002-2003)
  • Robert Dawson Evans Fellow Excellence in Teaching Award, Boston University School of Medicine, Department of Internal Medicine (2000)
  • House Officer Research Award, University of Michigan Hospitals, Department of Internal Medicine (1998)
View All 7honors and awards of Tushar Desai

Professional Education

Medical Education: Tufts University MA (1995)
Internship: University of Michigan Medical Center MI (1996)
Residency: University of Michigan Medical Center MI (1998)
Board Certification: Pulmonary Disease, American Board of Internal Medicine (2001)
Fellowship: Boston University School of Medicine MA (2002)
BA: Amherst College, Psychology (1991)
View All 7

Research & Scholarship

Current Research and Scholarly Interests

My lab is interested in understanding how alveolar epithelial type (AT) 1 and AT 2 cells are generated during lung development and replaced in adult life during aging and following injury. We use mouse genetic tools to specifically mark and fate-map AT 1 and AT 2 cells, in order to understand their differentiative potential and the lineage hierarchies that operate during alveolar epithelial turnover. By genetically deleting or mis-expressing transcription factors and other genes in AT 1 and AT 2 cells, we are also seeking to elucidate the specific role each gene plays in these cells and indirectly in the lung overall. We are interested not only in the role for AT 1 and AT 2 cells in health, but in exploring how their depletion or dysregulation may contribute to specific diseases, such as adenocarcinoma, emphysema, bronchopulmonary dysplasia, and fibrotic diseases of the lung.




Graduate and Fellowship Program Affiliations



Publication tag cloud

Publication Topics

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